Straddle Fracture
A straddle fracture refers to bilateral fractures of the superior and inferior pubic rami (i.e., fractures of all four pubic rami), creating a free-floating anterior pelvic segment.[1-2] The term d…
A straddle fracture refers to bilateral fractures of the superior and inferior pubic rami (i.e., fractures of all four pubic rami), creating a free-floating anterior pelvic segment.[1-2] The term derives from the classic mechanism of falling astride a hard object (e.g., fence, beam, bicycle crossbar), though it also occurs in high-energy trauma such as motor vehicle collisions and falls from height.[3-4] Because the pelvis is a ring structure, an isolated anterior ring fracture almost always implies a concomitant posterior ring injury — CT studies show posterior pelvic ring lesions in up to 96.8% of patients with pubic rami fractures.[5-6]
The following figure illustrates a comminuted pubic rami fracture with symphyseal offset, demonstrating the principle that displaced anterior ring fractures should prompt evaluation for a second disruption in the posterior ring:
1. History
- Mechanism: fall astride a hard object (classic "straddle" mechanism), MVC, motorcycle collision, pedestrian struck, fall from height, or crush injury[3]
- Pain location: groin, perineum, suprapubic region, low back, or hip
- Ability to ambulate or bear weight — inability to mobilize is a key concern for occult instability[8]
- Voiding symptoms: hematuria, inability to void, blood at urethral meatus (suggests urethral or bladder injury)[9]
- Timing: acute onset with trauma vs. insidious onset (consider insufficiency fracture in elderly/osteoporotic patients)[10]
- Anticoagulant use — increases hemorrhagic risk even in "stable" fractures[11]
2. Alarm Features
- Hemodynamic instability (SBP <90, HR >120): suggests significant pelvic hemorrhage — mortality up to 32% in unstable pelvic fractures[12]
- Blood at the urethral meatus, perineal butterfly hematoma, inability to void → suspect urethral injury[9]
- Gross hematuria → suspect bladder injury[9]
- Open fracture (perineal laceration, rectal/vaginal wound) — dramatically increases mortality and infection risk[13]
- Progressive abdominal distension or peritonitis → intraperitoneal bladder rupture or associated abdominal injury
- Neurologic deficits in lower extremities → lumbosacral plexus injury
- Delayed hemodynamic deterioration in elderly patients — corona mortis artery avulsion can present 48–72 hours after seemingly benign pubic rami fractures[11]
3. Medications
- Analgesics: Acetaminophen, NSAIDs (if no contraindication), opioids for acute pain management
- VTE prophylaxis: LMWH (enoxaparin 30 mg BID or 40 mg daily) is the most commonly used agent; aspirin (81 mg BID) shown noninferior to LMWH for prevention of death from any cause in the PREVENT CLOT trial. Initiate within 24 hours if hemodynamically stable[14-16]
- Anticoagulants to review: Patients on warfarin, DOACs, or antiplatelet agents are at increased hemorrhagic risk and may need reversal
- Avoid: Foley catheter insertion if urethral injury suspected — obtain retrograde urethrogram first[9]
4. Diet
- NPO if hemodynamically unstable or surgical intervention anticipated
- Adequate calcium and vitamin D supplementation in elderly/osteoporotic patients for long-term bone health[10]
- Ensure adequate hydration and nutrition to support fracture healing
- High-fiber diet or stool softeners to prevent straining during recovery
5. Review of Systems
- GU: Hematuria, dysuria, inability to void, blood at meatus, vaginal bleeding
- GI: Rectal bleeding (5% of urethral injuries have concomitant rectal injury), abdominal pain, distension[9]
- Neurologic: Lower extremity weakness, numbness, saddle anesthesia (lumbosacral plexus or cauda equina)
- Vascular: Signs of DVT (leg swelling, calf pain) — up to 34% develop proximal DVT[15]
- MSK: Low back pain (posterior ring injury), hip pain, inability to bear weight
6. Collateral History and Family History
- Witnesses to mechanism (speed, height of fall, position of impact)
- Pre-injury ambulatory status and functional baseline (especially in elderly)
- History of osteoporosis, prior fragility fractures, pelvic irradiation, rheumatoid arthritis, chronic corticosteroid use — all predispose to insufficiency fractures of the pubic rami[10][17]
- Anticoagulant/antiplatelet medication use
- Family history of osteoporosis or bleeding disorders
7. Risk Factors
- High-energy trauma: MVC, motorcycle collision, pedestrian struck, fall from height[3]
- Low-energy/insufficiency fractures: Osteoporosis, advanced age, female sex, rheumatoid arthritis, chronic corticosteroid use, pelvic irradiation, renal failure, post-hip surgery mechanical changes[10][17]
- Stress fractures: Long-distance runners, female athletes (pubic ramus stress fractures near symphysis)[18]
- Anticoagulation therapy increases hemorrhagic complications[11]
- Male sex increases risk of associated urethral injury (urethral injury in 6.1% of males vs. 0.5% of females with pelvic fractures)[19]
8. Differential Diagnosis
- Unstable pelvic ring disruption (APC-II/III, LC-II/III, vertical shear) — must rule out with CT[3][20]
- Acetabular fracture — found in 28.8% of patients with pubic rami fractures on CT[5]
- Hip fracture (femoral neck, intertrochanteric) — overlapping presentation with groin pain and inability to ambulate[21]
- Pubic symphysis diastasis ("open book" injury)
- Sacral fracture (often occult on plain films) — present in nearly all patients with pubic rami fractures[5-6]
- Insufficiency fracture of pubic rami in elderly — may mimic traumatic fracture[10]
- Pubic ramus stress fracture in athletes[18]
- Osteitis pubis or athletic pubalgia
9. Past Medical History
- Prior pelvic fractures or pelvic surgery
- Osteoporosis, osteopenia, metabolic bone disease
- Rheumatoid arthritis, chronic steroid use[17]
- Prior pelvic irradiation[10]
- History of DVT/PE
- Anticoagulant use
- Chronic kidney disease (affects bone quality)
10. Physical Exam
- Vitals: Tachycardia and hypotension suggest hemorrhage — pelvic fractures can cause massive retroperitoneal bleeding[12]
- Inspection: Perineal ecchymosis (butterfly hematoma), scrotal/labial swelling, blood at urethral meatus, open wounds[9]
- Palpation: Tenderness over pubic symphysis, pubic rami, sacroiliac joints; avoid repeated manual pelvic compression/distraction testing — it is neither sensitive nor specific and may worsen hemorrhage[22]
- Rectal/vaginal exam: Assess for open fracture, rectal tone, high-riding prostate (low sensitivity/specificity for urethral injury)[9]
- Neurovascular: Lower extremity motor/sensory exam, pedal pulses
- Functional: Ability to bear weight — inability to mobilize suggests occult instability in 42% of geriatric patients with seemingly isolated rami fractures[8]
11. Lab Studies
- CBC: Serial hemoglobin/hematocrit to monitor for hemorrhage
- Type and screen/crossmatch: Essential in unstable fractures; activate massive transfusion protocol if hemodynamically unstable[15][23]
- Coagulation studies: PT/INR, PTT (especially if on anticoagulants)
- BMP/CMP: Renal function, electrolytes
- Lactate: Marker of tissue hypoperfusion/occult hemorrhage
- Urinalysis: Hematuria screening — present in most bladder injuries[9]
- Anti-Factor Xa levels: If on LMWH, to ensure adequate prophylactic dosing (many pelvic fracture patients are underdosed)[16]
12. Imaging
- AP pelvis radiograph: First-line screening; identifies pubic rami fractures, symphyseal diastasis, and gross displacement. However, posterior ring injuries are frequently occult on plain films[5][20-21]
- CT pelvis with contrast: Gold standard for full characterization of pelvic ring injury, detection of posterior ring fractures (sacral fractures, SI joint disruption), acetabular fractures, and active hemorrhage (contrast extravasation)[15][24]
- Retrograde urethrogram (RUG): Indicated before Foley placement if blood at meatus or suspected urethral injury[9][25]
- CT cystogram: If bladder injury suspected (gross hematuria with pelvic fracture)[9]
- MRI: Useful for detecting occult posterior ring injuries (sacral insufficiency fractures) not visible on CT, particularly in geriatric patients who fail to mobilize[8]
- Bone scintigraphy: Historically used for occult fractures; largely replaced by MRI[10]
13. Special Tests
- Young-Burgess Classification: Categorizes pelvic ring injuries by mechanism — APC (I–III), LC (I–III), vertical shear, combined mechanism. Useful for predicting instability, transfusion needs, and guiding surgical decision-making[20][26]
- Tile/AO Classification: Type A (stable), Type B (rotationally unstable), Type C (rotationally and vertically unstable)[2]
- FAST/E-FAST: Rapid bedside assessment for intraabdominal free fluid and pubic symphyseal widening[15][27]
- Stress radiographs under fluoroscopy: Can identify dynamic instability in patients with seemingly isolated rami fractures who fail to mobilize[8]
- Diagnostic peritoneal aspiration (DPA): Alternative to FAST in hemodynamically unstable patients to rule out intraabdominal hemorrhage[15]
14. ECG
- Not specific to straddle fractures, but should be obtained in:
- Elderly patients (baseline cardiac assessment)
- Polytrauma patients
- Patients with hemodynamic instability to rule out cardiac causes
- Pre-operative assessment
- Monitor for signs of right heart strain (S1Q3T3, new RBBB) if PE suspected in the setting of VTE
15. Assessment
- A straddle fracture represents a bilateral anterior pelvic ring disruption that, by the ring principle, almost always involves a concomitant posterior injury.[5-6] Severity ranges widely:
- Low-energy (elderly/insufficiency): Often minimally displaced, may appear benign but carries significant morbidity — 16% 90-day mortality in geriatric patients with rami fractures and inability to mobilize. Occult posterior ring instability is present in ~42%[8]
- High-energy (trauma): Associated with life-threatening hemorrhage (mortality up to 32%), genitourinary injuries (4.5–7.7%), and polytrauma[12][19][28]
- Complications: hemorrhage, urethral/bladder injury, DVT/PE, infection (open fractures), chronic pain, sexual dysfunction, malunion/nonunion
16. Treatment Plan
Initial stabilization (ED)
- ATLS primary survey; apply pelvic binder at the level of the greater trochanters with legs internally rotated[15][22-23]
- If hemodynamically unstable: activate massive transfusion protocol (1:1:1 pRBC:FFP:platelets or whole blood), consider TXA[23]
- Establish urinary drainage — if no signs of urethral injury, place Foley; if urethral injury suspected, obtain RUG first → suprapubic catheter for complete PFUI[9][29]
Hemorrhage control (unstable patients)
- FAST/DPA to rule out intraabdominal hemorrhage[15]
- If intraabdominal hemorrhage → OR for laparotomy + consider external fixation
- If no intraabdominal source → pelvic external fixation + preperitoneal packing → angioembolization if still unstable[15]
- REBOA may be considered in extremis[12][23]
Definitive management
- Stable fractures (APC-I, LC-I): Conservative management with weight-bearing as tolerated, pain control, early mobilization, and physical therapy[2-3]
- Unstable fractures (APC-II/III, LC-II/III, VS): Surgical fixation — anterior plating of symphysis for diastasis >2.5 cm; retrograde transpubic screws or anterior plating for straddle fractures; posterior fixation as indicated[1][3]
- VTE prophylaxis: Initiate LMWH or aspirin as soon as hemodynamically stable, ideally within 24 hours[14-16]
17. Disposition
- Admit (ICU or monitored bed): Hemodynamic instability, need for transfusion, unstable fracture pattern, associated injuries (urethral, bladder, vascular), polytrauma, inability to mobilize, need for surgical fixation[30-31]
- Admit (floor): Stable straddle fracture with inability to bear weight, elderly patients requiring pain management and PT evaluation, patients on anticoagulation requiring monitoring
- Observation: Hemodynamically stable patients with minimally displaced fractures who need serial hemoglobin checks and functional assessment
- Discharge (rare for true straddle fractures): Only if minimally displaced, hemodynamically stable, able to ambulate with assistive device, adequate pain control, reliable follow-up, and no GU injury
- Consult triggers: Orthopedic surgery (all pelvic ring injuries), urology (blood at meatus, hematuria, voiding difficulty), interventional radiology (active hemorrhage on CT), trauma surgery (polytrauma, hemodynamic instability)[30]
18. Follow Up / Return Precautions
- Orthopedic follow-up: Within 1–2 weeks with repeat imaging to assess alignment and healing
- Urology follow-up: Essential for all patients with urethral or bladder injury — timing of definitive urethroplasty typically 3–6 months post-injury[32]
- VTE prophylaxis duration: Continue for up to 4 weeks post-injury for high-risk patients; consider extended prophylaxis with aspirin or LMWH post-discharge[33]
- Return precautions: Instruct patients to return immediately for increasing pain, inability to urinate, blood in urine, fever, leg swelling, shortness of breath, lightheadedness/syncope, or worsening inability to bear weight
- Expected recovery: Stable fractures typically heal in 6–12 weeks with conservative management; elderly patients may have prolonged recovery and increased 90-day mortality (16%). Stress fractures in athletes require cessation of running until symptom-free[8][18]
- Long-term considerations: Monitor for chronic pelvic pain, sexual dysfunction, urethral stricture (in PFUI patients), and post-traumatic arthritis

References
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